Skip to main content

Publications

Included here is a list of publications from OCG programs. All published data are available to the research community through the program-specific data matrices.

* denotes publications from the CTD2 initiative that are results of intra-Network collaborations

 

CTD²
Cancer Research

Investigators use a high-throughput assay to identify genomic predictors of radiation sensitivity

CTD²
Cancer Cell

Researchers document coexpression of EGFR and EGFRvIII in primary human glioblastoma that drives transformation and tumorigenesis in a cell-intrinsic manner and clarify specific oncogenic signaling relationships in glioblastoma.

CTD²
Clinical Cancer Research

Author comment on Dual blockade of the PI3K/AKT/mTOR(AZD8055) and RAS/MEK/ERK (AZD6244) pathways synergystically inhibits rhabdomyosarcoma... 

CTD²
Cold Spring Harbor Perspective in Medicine

A review on the role of MYCN and other newly identifed drivers in neuroblastoma.

CTD²
Nature Genetics

A collaborative effort to compare the molecular changes across the original 12 tumor types profiled by The Cancer Genome Atlas.

CTD²
Journal of the American Chemical Society

Using a newly developed platform to identify the signaling pathway/molecular target of natural products, we identified a family of alkaloid natural products, discoipyrroles A-D (1-4), from Bacillus hunanensis that inhibit the DDR2 signaling pathway. 

CTD²
Cancer Discovery

3q26 is frequently amplified in several cancer types with a common amplified region containing 20 genes. To identify cancer driver genes in this region, we interrogated the function of each of these genes.

CTD²
Cell

The Cancer Therapeutics Response Portal (www.broadinstitute.org/ctrp) was created to identify cancer genotype-compound sensitivity relationships.

CGCI
Blood

By utilizing whole-genome sequencing, DNA copy number analysis and RNA-seq, researchers discovered recurrent somatic point mutations and genes that were targeted by focal somatic deletions in diffuse large B-cell lymphoma (DLBCL).